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Mitchell AP: Function of Candida albicans Adhesin Hwp1 in Biofilm Formation. Eukaryot Cell 2006, 5:1604–1610.PubMedCentralPubMedCrossRef 39. Ramage G, Vande Walle K, Wickes BL, López-Ribot JL: Standardized method for in vitro antifungal susceptibility testing of Candida albicans biofilms. Antimicrob Agents Chemother 2001, 45:2475–2479.PubMedCentralPubMedCrossRef 40. Chandra J, Mukherjee PK, Ghannoum MA: In vitro growth and analysis of Candida biofilms. Nat Protoc 2008, 3:1909–1924.PubMedCrossRef Competing interests The authors declare that they have no competing interests. Authors’ contributions LY2228820 concentration XRD conceived and

designed the experiments and carried out most of the data collection and drafted the manuscript. ZHZHL participated in data analysis and interpretation and drafted the manuscript. JRS conceived the study, participated in its design and revised the manuscript. DHY contributed to data analysis. All authors read and approved the final manuscript.”
“Background Pseudomonas syringae comprises a large and well-studied group of plant-pathogenic bacteria [1]. They infect a broad range of host plants and are subdivided into more than 50 different pathogenic variants called pathovars [2]. P. syringae possesses a number of well-studied virulence and pathogenicity factors such as the Type III effector trafficking system, various phytotoxins, different Chlormezanone mechanisms suppressing the plant defense, or synthesis of exopolysaccharides [3–5]. Exopolysaccharides play a variety of roles in virulence and pathogenicity not only in Pseudomonas but also in other biofilm-producing organisms [6, 7]. The two major exopolysaccharides produced by P. syringae pv. glycinea are alginate and levan [7]. Levan is a β-(2,6) polyfructan with extensive branching through β-(2,1) linkages, while alginate is a copolymer of O-acetylated β-(1,4)-linked D-mannuronic acid and its C-5 epimer, L-guluronic acid [7–10]. P. syringae pv.

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